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ANP activation of the ANP catalytic receptor will stimulate its intracellular guanylyl cyclase activity to convert GTP to cGMP. cGMP will then stimulate cGMP-dependent protein kinase (PKG), which will then induce smooth muscle relaxation. This is particularly important in the vasculature, where vascular smooth muscle will bind ANP released as a result of increasing right atrial pressure and will cause the walls of the vasculature to relax. This relaxation will decrease total peripheral resistance, which will in turn decrease venous return to the heart. The decrease in venous return to the heart will reduce the preload and will result in the heart's having to do less work.

NPR

货号 产品名 CAS号 信息
CSN21949 Brain Natriuretic Peptide (1-32), Human 114471-18-0 Brain Natriuretic Peptide (1-32), Human is an endogenous peptide agonist at ANP receptor A (NPR1).
CSN32968 NPR1 Agonist 10 2562321-74-6 Atrial Natriuretic Peptide A (NPR1; Guanylate Cyclase A) Receptor Agonists Signal Transduction Modulators
CSN32978 NPR1 Agonist 19 2143955-64-8 Atrial Natriuretic Peptide A (NPR1; Guanylate Cyclase A) Receptor Agonists Signal Transduction Modulators
CSN32959 NPR1 Agonist 5 2144388-77-0 Atrial Natriuretic Peptide A (NPR1; Guanylate Cyclase A) Receptor Agonists Signal Transduction Modulators
CSN32979 NPR1 Agonist 20 2147721-31-9 Atrial Natriuretic Peptide A (NPR1; Guanylate Cyclase A) Receptor Agonists Signal Transduction Modulators
CSN32970 NPR1 Agonist 12 2562327-68-6 Atrial Natriuretic Peptide A (NPR1; Guanylate Cyclase A) Receptor Agonists Signal Transduction Modulators
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